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Table representation of search results timeline featuring number of search results per year.
Year | Number of Results |
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2002 | 1 |
2008 | 1 |
2011 | 1 |
2024 | 0 |
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The potential use of AKAP18delta as a drug target in heart failure patients.
Expert Opin Biol Ther. 2008 Aug;8(8):1099-108. doi: 10.1517/14712598.8.8.1099.
Expert Opin Biol Ther. 2008.
PMID: 18613762
Review.
Calcium reabsorption is essential for relaxation and filling of the heart and is the rate-limiting step for making the heart beat faster in response to adrenaline or noradrenaline. We conclude that targeting AKAP18delta may have application in manipulating calcium r …
Calcium reabsorption is essential for relaxation and filling of the heart and is the rate-limiting step for making the heart b …
Deletion of the distal C terminus of CaV1.2 channels leads to loss of beta-adrenergic regulation and heart failure in vivo.
Fu Y, Westenbroek RE, Yu FH, Clark JP 3rd, Marshall MR, Scheuer T, Catterall WA.
Fu Y, et al.
J Biol Chem. 2011 Apr 8;286(14):12617-26. doi: 10.1074/jbc.M110.175307. Epub 2011 Jan 7.
J Biol Chem. 2011.
PMID: 21216955
Free PMC article.
L-type calcium currents conducted by CaV1.2 channels initiate excitation-contraction coupling in cardiac and vascular smooth muscle. In the heart, the distal portion of the C terminus (DCT) is proteolytically processed in vivo and serves as a noncovalently associated autoi …
L-type calcium currents conducted by CaV1.2 channels initiate excitation-contraction coupling in cardiac and vascular smooth muscle. In the …
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Contributions of protein kinase A anchoring proteins to compartmentation of cAMP signaling in the heart.
Kapiloff MS.
Kapiloff MS.
Mol Pharmacol. 2002 Aug;62(2):193-9. doi: 10.1124/mol.62.2.193.
Mol Pharmacol. 2002.
PMID: 12130668
Review.
The cAMP-dependent protein kinase (PKA) transduces signals in the heart initiated by beta(1)-adrenergic, G-protein-coupled receptors after norepinephrine, sympathetic stimulation. ...
The cAMP-dependent protein kinase (PKA) transduces signals in the heart initiated by beta(1)-adrenergic, G-protein-coupled receptors …
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